> Not neccesarily. There are actually two kinds of
> Cholesterol. There is HDL, the "good"
> Cholestrol and there is LDL, the "bad"
> Cholesterol.
> Too much LDL cholesterol can clog your arteries,
> increasing your risk of heart attack and stroke.
> Your body makes HDL cholesterol for your protection. It
> carries cholestrol away from your arteries.
> Studies suggest that high levels of HDL cholesterol
> reduce your risk of heart attack.
Conterary to beliefs, that's really not true.
People may think that LDL cholestral are the artery clogging cholestrol that you produce.
New studies shown that there is a minimum of LDL cholestral, and your HDL should make no less than 25 percent but no more than 40 percent of your total cholestral. A healthy total cholestral range is actually between 190 and 250.
When you take in saturated fatty acids, your liver produces LDL cholestral molecues to deliver the proteins and vitamins towards every parts of your body. Your HDL cholestral is designed to keep your arteries clean that your LDL has left behind. Did you know that cholestral is actually protein surrounded by fat? That's true.
New studies suggest that it's your tryglycerides and your Lp, NOT your LDL, that are the "true" bad cholestrol.
But watch out. Trans fats are still the killers. How your trans fats raise your LDL and lower your HDL? Trans fats damages your liver's function and causes it to "malfunction" and overproduce LDL cholestral, while it blocks the ability to produce HDL cholestral. It also makes the liver produce VLDL cholestral, which is more likely to stick to arteries. That's why Trans fats can cause fat to build up in your liver.
Coconut oil, on the other hand, is food for your liver, your organs, and even your heart. It regulates your HDL and your LDL cholestral, act as emzymes, and even can aid in better repair towards your organs. Your liver can also normally produce the proper LDL again to deliver the vitamins, minierals, and protein as it should, as well as the HDL cholestral to keep the arteries clean and from overplaquing the damaged part of the artery.